By Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)
In this age once we are witnessing a veritable explosion in new modalities in diagnos tic imaging we proceed to have a very good want for precise experiences of the vascularity of the mind in sufferers who've every kind of cerebral vascular sickness. a lot of the knowledge of cerebral vascular occlusive lesions which we constructed within the final twenty years was once in line with our skill to illustrate the vessels that have been affected. a lot experimental paintings in animals have been performed the place significant cerebral vessels have been obstructed and the consequences of those obstructions at the mind saw pathologically. besides the fact that, it used to be now not till cerebral angiography can be played with the element that turned attainable within the many years of the '60 's and for that reason that lets start to comprehend the connection of the obstructed vessels saw angiographically to the medical findings. furthermore, a lot physiologic info was once bought. for example, the idea that ofluxury perfusion that's used to explain non-nutritional circulation in the course of the tissues was once saw first angiographically even supposing the time period used to be now not used till LASSEN defined it as a pathophysiological phenomenon saw in the course of cerebral blood circulate experiences with radioactive isotopes. the idea that of embolic occlusions of the cerebral vessels as opposed to thrombosis used to be clarified and the relative frequency of thrombosis as opposed to embolism used to be greater understood. the concept that of collateral flow of the mind via so-called meningeal end-to finish arterial anastomoses was once significantly greater understood while serial angiography in obstructive cerebral vascular sickness used to be performed with expanding frequency.
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Additional info for Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases
Is now more recognizable; filling of the carotid siphon and of the intracranial vessels (recanalization of an occluded carotid artery) 39 , T T a Fig. 10a-c. Subocclusion: a Large plaque (~) on the carotid bifurcation, which has almost completely occluded the left internal carotid artery. Above the plaque the lumen of the vessel is filled by a thrombus (_). Further cranially, the internal carotid artery is small and the filling is delayed, but the lumen seems to be free. ) and a direct filling 40 b of the carotid siphon from branches of the external carotid artery (~).
Kinking is a deformation typically occurring in aged arteries. In a recent radio anatomic study CLARISSE et al. (1979) showed that with advancing age dilatation and elongation of the artery due to thinning of the wall and to a loss of elasticity, respectively, occur. The consequences of this process are different grades of coiling or kinking. The stenosis in cases of kinking can be purely mechanical or due to additional arteriosclerotic plaques. A thrombus can originate there and lead to occlusion of the artery (Fig.
A careful description of all lesions is mandatory since in some cases only the multiplicity of lesions gives a clue to the symptomatology of the patient (Figs. 17, 18, 22, 23). On the other hand, lesions may be present and sometimes may also be very extensive without corresponding clinical symptoms (Figs. 14, 15, 16,23). 5 Tortuosity In accordance with the description of WEIBEL and FIELDS (1965a, b, 1969) the elongation of the internal carotid artery is classified according to the following groups: Tortuosity: S- or C-shaped elongation Coiling: Exaggerated S-shaped or circular curve (Fig.
Angiography and Computed Tomography in Cerebro-Arterial Occlusive Diseases by Professor Dr. Gianni Boris Bradač, Priv.-Doz. Dr. Roland Oberson (auth.)